Type 2 Diabetes - The Feel-Full Hormone That Affects Your Weight!
Food dramatically impacts people with Type 2 diabetes in many different ways...
some which are good, and some which are not so good.
But one compound in particular impacts diabetes more than you realize.
Most people, including Type 2 diabetics, have never even heard of it, know what it is or what role it plays within their body.
The compound is leptin.
Leptin is one of several hormones your body makes that tells you to stop eating.
Leptin is a protein hormone made up of some 167 amino acids and is formed from adipose tissue.
The amount of leptin present in a person's body is directly determined by how much fat is also in their body.
Insulin is used to transport sugar to your cells to be used for energy.
The sugar can come from simple sugar or food broken down into sugar.
Some of this sugar is deposited into your adipose tissue, where it releases leptin.
The leptin hormone which is then secreted by your fat cells, is sent to the hypothalamus gland in your brain.
Leptin then tells your brain how much fat is being stored from your meal.
As we continue to eat, we continue making insulin which continues producing leptin.
When enough leptin is measured, your brain is notified your body has had enough to eat and to stop making insulin and start burning fat.
This is the full sensation we feel after a meal.
If we continue to eat, the insulin that was previously transporting sugar to cells changes jobs and begins to cause the body to begin producing triglycerides in order to deal with the surplus amount of sugar.
At the same time, the production of triglycerides interferes with the transportation and detection of leptin in the brain.
This is why, once you pass the "shut-off" point, your brain loses it's ability to inform you to stop eating.
At a certain point, your leptin levels will continue to climb because it's ability to signal your brain has been hampered.
When leptin has a hard time delivering its message to your brain, a condition known as leptin resistance has developed.
The entire process can be summed up like this: your brain cannot be notified your body has eaten enough food because the process of leptin delivery has been interfered with.
So, you continue to eat and ultimately gain weight.
The problem is, unless you manage to sort this process out this cycle will continue to occur.
If you are overweight, you are therefore leptin resistant.
Your brain and pancreas simply cannot hear the leptin signal.
Your body thinks you are in starvation mode even if you are still eating.
Leptin resistance occurs years before insulin resistance and can be measured via a blood test.
some which are good, and some which are not so good.
But one compound in particular impacts diabetes more than you realize.
Most people, including Type 2 diabetics, have never even heard of it, know what it is or what role it plays within their body.
The compound is leptin.
Leptin is one of several hormones your body makes that tells you to stop eating.
Leptin is a protein hormone made up of some 167 amino acids and is formed from adipose tissue.
The amount of leptin present in a person's body is directly determined by how much fat is also in their body.
Insulin is used to transport sugar to your cells to be used for energy.
The sugar can come from simple sugar or food broken down into sugar.
Some of this sugar is deposited into your adipose tissue, where it releases leptin.
The leptin hormone which is then secreted by your fat cells, is sent to the hypothalamus gland in your brain.
Leptin then tells your brain how much fat is being stored from your meal.
As we continue to eat, we continue making insulin which continues producing leptin.
When enough leptin is measured, your brain is notified your body has had enough to eat and to stop making insulin and start burning fat.
This is the full sensation we feel after a meal.
If we continue to eat, the insulin that was previously transporting sugar to cells changes jobs and begins to cause the body to begin producing triglycerides in order to deal with the surplus amount of sugar.
At the same time, the production of triglycerides interferes with the transportation and detection of leptin in the brain.
This is why, once you pass the "shut-off" point, your brain loses it's ability to inform you to stop eating.
At a certain point, your leptin levels will continue to climb because it's ability to signal your brain has been hampered.
When leptin has a hard time delivering its message to your brain, a condition known as leptin resistance has developed.
The entire process can be summed up like this: your brain cannot be notified your body has eaten enough food because the process of leptin delivery has been interfered with.
So, you continue to eat and ultimately gain weight.
The problem is, unless you manage to sort this process out this cycle will continue to occur.
If you are overweight, you are therefore leptin resistant.
Your brain and pancreas simply cannot hear the leptin signal.
Your body thinks you are in starvation mode even if you are still eating.
Leptin resistance occurs years before insulin resistance and can be measured via a blood test.
